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ニュース @wikiのwikiモードでは #news(興味のある単語) と入力することで、あるキーワードに関連するニュース一覧を表示することができます 詳しくはこちらをご覧ください。 =>http //atwiki.jp/guide/17_174_ja.html たとえば、#news(wiki)と入力すると以下のように表示されます。 ウィキペディアを作ったiMacが箱付きで競売に登場。予想落札価格は約96万円!(ギズモード・ジャパン) - Yahoo!ニュース - Yahoo!ニュース メトロイド ドレッド攻略Wiki - Gamerch(ゲーマチ) ツムツム攻略Wiki|ゲームエイト - Game8[ゲームエイト] 【グランサガ】リセマラ当たりランキング - グランサガ攻略wiki - Gamerch(ゲーマチ) アイプラ攻略Wiki|アイドリープライド - AppMedia(アップメディア) Among Us攻略Wiki【アマングアス・アモングアス】 - Gamerch(ゲーマチ) マニュアル作成に便利な「画像編集」機能を提供開始! - ナレッジ共有・社内wikiツール「NotePM」:時事ドットコム - 時事通信 マニュアル作成に便利な「画像編集」機能を提供開始! - ナレッジ共有・社内wikiツール「NotePM」 - PR TIMES 【Apex Legends】ヴァルキリーの能力と評価【エーペックス】 - Gamerch(ゲーマチ) モンハンライズ攻略Wiki|MHRise - AppMedia(アップメディア) ポケモンBDSP(ダイパリメイク)攻略wiki - AppMedia(アップメディア) SlackからWikiへ!シームレスな文章作成・共有が可能な「GROWIBot」リリース - アットプレス(プレスリリース) 【ウマ娘】チャンピオンズミーティングの攻略まとめ - Gamerch(ゲーマチ) 【ウマ娘】ナリタブライアンの育成論|URAシナリオ - Gamerch(ゲーマチ) 【ウマ娘】ヒシアケボノの育成論|URAシナリオ - Gamerch(ゲーマチ) 【ウマ娘】カレンチャンの育成論|URAシナリオ - Gamerch(ゲーマチ) 【ウマ娘】フジキセキの育成論|URAシナリオ - Gamerch(ゲーマチ) ドラゴンクエストけしケシ攻略Wiki - Gamerch(ゲーマチ) 【スタオケ】カード一覧【金色のコルダスターライトオーケストラ】 - Gamerch(ゲーマチ) 【スマブラSP】ソラのコンボと評価【スマブラスペシャル】 - Gamerch(ゲーマチ) 【ブレフロレゾナ】リセマラ当たりランキング【ブレイブフロンティアレゾナ】 - ブレフロR攻略Wiki - Gamerch(ゲーマチ) 【ポケモンユナイト】サーナイトの評価と性能詳細【UNITE】 - Gamerch(ゲーマチ) 仲村トオル、共演者は事前に“Wiki調べ”(オリコン) - Yahoo!ニュース - Yahoo!ニュース 【ENDER LILIES】攻略チャートと全体マップ【エンダーリリィズ】 - Gamerch(ゲーマチ) 【ウマ娘】あんしん笹針師の選択肢はどれを選ぶべき? 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- AUTOMATON スタスマ攻略Wiki【スタースマッシュ】 - Gamerch(ゲーマチ) 無料とは思えない多機能っぷりなWikiインフラ「Wiki.js」レビュー、自前でホスト&外部サービスと連携可能 - GIGAZINE Microsoft Teamsの基本と活用(24) TeamsのWikiを使う - マイナビニュース 『ゲーミングお嬢様』での提起が話題に “企業系wiki”に横たわる問題点とは - リアルサウンド 「エイリアンのたまご」,自動周回機能と公式wikiが登場 - 4Gamer.net 【リゼロス】Re ゼロから始める異世界生活 Lost in Memories攻略まとめwiki - 電撃オンライン 【世界初!】モノの背景を全方位で執筆できるVintage Wiki「VOV」を正式リリース - PR TIMES プロジェクトセカイ攻略Wiki【プロセカ】 - Gamerch(ゲーマチ) パワプロ2021/2020攻略Wiki - Gamerch(ゲーマチ) ヌーラボ、「Backlog」の絵文字入力の補完機能やWiki編集の自動マージ機能を改善 - CodeZine(コードジン) ヌーラボ、プロジェクト管理ツール「Backlog」の絵文字入力の補完機能・Wiki編集の自動マージ機能を修正改善 - PR TIMES Backlog、Wikiにファイル添付が容易にできる機能をリリース -- グローバルバーの視認性改善なども実施 - PR TIMES GK川島、パンチング失点でWiki書き換え炎上 「セネガル代表」「プロボクサー」... - J-CASTニュース
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TOXICOLOGICAL PROFILE FOR WHITE PHOSPHORUS -index ▼2 HEALTH EFFECTS ▼▼2.2 DISCUSSION OF HEALTH EFFECTS BY ROUTE OF EXPOSURE 2.2.2 Oral Exposure -2 2.2.2 Oral Exposure(2.2.2.0 preface) 2.2.2.1 Death 2.2.2 Oral Exposure -22.2.2.2 Systemic Effects(preface) Respiratory Effects. Cardiovascular Effects. Gastrointestinal Effects. Hematological Effects. Musculoskeletal Effects. Hepatic Effects. Renal Effects. Dermal Effects. Other Systemic Effects. 2.2.2 Oral Exposure -32.2.2.3 Immunological and Lymphoreticular Effects 2.2.2.4 Neurological Effects 2.2.2.5 Reproductive Effects 2.2.2.6 Developmental Effects 2.2.2.7 Genotoxic Effects 2.2.2.8 Cancer 2.2.2.2 Systemic Effects (preface) Systemic effects of white phosphorus in humans and animals after oral exposure are discussed below. The highest NOAEL value and all reliable LOAEL values from each reliable study for systemic effects in each species and duration category are recorded in Table 2-2 and plotted in Figure 2-2. No studies were located regarding ocular effects in humans or animals after oral exposure to white phosphorus. Respiratory Effects. Studies on respiratory effects following acute oral exposure of humans to white phosphorus were limited to case reports of intentional or accidental consumption of materials containing white phosphorus. Although intake of phosphorus was often reported, dose could be estimated for only one study (Hann and Veale 1910), because vomiting and/or gastric lavage nearly always occurred soon after poisoning, expelling much of the ingested phosphorus from the body. Tachypnea (increased respiratory rate; 48 breaths/minute) was observed in a woman consuming rat poison containing 4% white phosphorus (Hann and Veale 1910); the woman apparently did not vomit until the second day, and the vomitus was clear. The estimated dose was 2 mg/kg. Four days after ingesting the rat poison, the woman died, apparently from liver failure. Autopsy showed that the pleural cavity was filled with a dark fluid, but no histological abnormalities were observed in the lungs (Hann and Veale 1910). In the following studies, no doses could be estimated for respiratory effects because of vomiting and/or gastric lavage. In a case report involving ingestion of rat poison containing white phosphorus, the patient arrived at a hospital in a coma and displayed Cheyne-Stokes respirations and rales (Wechsler and Wechsler 1951). The Cheyne-Stokes respirations increased to an extreme degree, and the patient died. Autopsy revealed pulmonary congestion and edema throughout the stroma. Increased respiratory rate (56 breaths/minute) and rales also were observed in an infant ingesting rat poison containing white phosphorus (Rao and Brown 1974). The child died, and autopsy revealed evidence of pulmonary edema. Rales were observed in a child ingesting a fatal dose of white phosphorus in fireworks; autopsy indicated that the lungs were normal except for some fibrous adhesions (Dwyer and Helwig 1925). Hemorrhagic bronchopneumonia was observed following autopsy of a man ingesting a fatal dose of rat poison containing white phosphorus (Winek et al. 1973). Autopsy of a child who died following ingestion of a firecracker revealed fatty deposition in parenchyma, bronchial epithelium, and tracheal epithelium and cartilage (Humphreys and Halpert 1931). Death from cardiopulmonary failure was reported for a 63-year-old woman (Winek et al. 1973), a 2-year-old boy (Simon and Pickering 1976), and a 3-year-old girl (Simon and Pickering 1976) following ingestion of white phosphorus in rat poison; a respiratory rate of 44 breaths/minute was initially observed in the girl (Simon and Pickering 1976). Increased respiratory rate was observed prior to death in two case reports involving ingestion of rat poison (Talley et al. 1972; Winek et al. 1973). Shallow respirations and cyanosis were observed prior to death in an adult female following ingestion of rat/roach poison containing white phosphorus (Rubitsky and Myerson 1949). Rales were observed 1 day after intentional ingestion of rat poison by a 30-year-old man; 2 days later the patient went into shock but survived the poisoning and eventually recovered (Pietras et al. 1968). No treatment-related respiratory effects were reported in children treated with white phosphorus for intermediate durations. No treatment-related microscopic changes were observed in the lungs of rats exposed to 0.2 mg/kg/day white phosphorus in the diet for a chronic duration (Fleming et al. 1942) or 0.075 mg/kg/day phosphorus by gavage for an intermediate duration (LRDC 1985). Heavy breathing and apnea were reported following ingestion of a fatal quantity of white phosphorus by a cat (Frye and Cucuell969). Necropsy revealed hyperemia, hemorrhage and edema in the lungs. Cardiovascular Effects. Alterations in electrocardiograms, such as altered or inverted T waves and changes in the QRS complex, and other cardiac changes, such as tachycardia, arrhythmias, atrial fibrillation, and decreased ventricular contractility, have been observed in individuals accidentally or intentionally ingesting a single dose of white phosphorus (Dathe and Nathan 1946; Diaz-Rivera et al. 1950, 1961; Dwyer and Helwig 1925; Ehrentheil 1957; Matsumoto et al. 1972; McCarron et al. 1981; Newburger et al. 1948; Pietras et al. 1968; Rao and Brown 1974; Simon and Pickering 1976; Talley et al. 1972). Damage to the myocardium was verified by a number of cases in which histological examination of the heart was performed. Prominent cross striations in the myocardium (Dwyer and Helwig 1925), fatty infiltration of muscle (Diaz-Rivera et al. 1961; Humphreys and Halpert 1931; Wertham 1932), necrosis of myocardium (Wechsler and Wechsler 1951), markedly dilated cardiac chamber (Rao and Brown 1974), and interstitial edema of the myocardium and vacuolation of cells (Talley et al. 1972) have been observed. Because of vomiting and gastric lavage, doses cannot be calculated from the human studies. No cardiac effects were reported in longer term human studies. In addition to the effects on the heart, a number of vascular effects have been observed in humans acutely exposed to white phosphorus. A markedly decreased or undetectable blood pressure (Caley and Kellock 1955; Dathe and Nathan 1946; McCarron et al. 1981; Rubitsky and Myerson 1949; Simon and Pickering 1976; Wechsler and Wechsler 1951), vascular collapse (Diaz-Rivera et al. 1950, 1961), undetectable or decreased pulse (Dwyer and Helwig 1925; Rubitsky and Myerson 1949), and increased pulse (Dathe and Nathan 1946; Hann and Veale 1910; McCarron et al. 1981; Wechsler and Wechsler 1951) have been observed. In addition, individuals have died following cardiopulmonary arrest (Simon and Pickering 1976; Winek et al. 1973), which may be due to effects on the heart or vascular system. A dose of 2 mg/kg/day for vascular effects was identified from the Hann and Veale (1910) report of a woman ingesting a single dose of white phosphorus. Dose levels cannot be estimated for the other case reports. Hemorrhaging in internal organs, as well as the appearance of petechial hemorrhages on the skin, have been reported in a number of acute human exposure cases (Hann and Veale 1910; Humphreys and Halpert 1931; Winek et al. 1973). It is not known whether these effects are due to impairment of the integrity of the blood vessels or due to damage of the affected organ (e.g., liver, stomach) itself. In rats administered 0.075 mg/kg/day white phosphorus for an intermediate duration, no histological alterations were observed in the heart (Bio/dynamics 1991; IRDC 1985). In rats exposed for an intermediate duration to an unknown concentration of airborne white phosphorus from the furnace room of a phosphorus factory, an increase in permeability of capillary walls, lesions in the walls of blood vessels and evidence of impaired microcirculation were observed in the mouth (Ruzuddinov and Rys-Uly 1986). These effects probably resulted from the local action of white phosphorus on the oral cavity. Gastrointestinal Effects. Most of the human case reports listed vomiting as an early effect following ingestion of a single high dose of white phosphorus (Caley and Kellock 1955; Dathe and Nathan 1946; Diaz-Rivera et al. 1950; Dwyer and Helwig 1925; Ehrentheil 1957; Fletcher and Galambos 1963; Greenberger et al. 1964; Hann and Veale 1910; Humphreys and Halpert 193 1; Matsumoto et al. 1972; McCarron et al. 1981; McIntosh 1927; Newburger et al. 1948; Pietras et al. 1968; Rubitsky and Myerson 1949; Simon and Pickering 1976; Wechsler and Wechsler 1951; Winek et al. 1973). The doses that induced vomiting ranged from 2 to 23 mg/kg (Caley and Kellock 1955; Ehrentheil 1957; Fletcher and Galambos 1963; Harm and Veale 1910; Matsumoto et al. 1972; McCarron et al. 1981; Newburger et al. 1948; Rubitsky and Myerson 1949). Vomiting generally started within hours after ingesting the white phosphorus, and sometimes continued for many days. Other gastrointestinal effects included abdominal cramps or pain (often severe) (Dwyer and Helwig 1925; Ehrentheil1957; Fletcher and Galambos 1963; Greenberger et al. 1964; Humphreys and Halpert 1931; McCarron et al. 1981; Newburger et al. 1948; Pietras et al. 1968), vomiting blood and/or pieces of the gastric mucosa (Dathe and Nathan 1946; Diaz-Rivera et al. 1950; Rubitsky and Myerson 1949), necrosis and erosion of mucosa in the esophagus, stomach, duodenum, and jejunum (Wechsler and Wechsler 1951), and gastrointestinal hemorrhage (Dwyer and Helwig 1925; Hann and Veale 1910; Humphreys and Halpert 1931; Wertham 1932, Winek et al. 1973). These effects, with the exception of necrosis, were probably due to the irritating effects of white phosphorus on the mucosa of the gastrointestinal tract. Vomitus often contained white phosphorus, indicating that vomiting generally occurred before all white phosphorus and/or its oxidation products had been absorbed. No gastrointestinal effects were reported in children receiving treatment with 0.026-0.158 mg/kg/day white phosphorus for as much as 26 months (Phemister 1918; Compere 1930a). An infant became seriously ill during treatment with 0.083 mg/kg/day white phosphorus (6-month time-weighted average dose), but recovered entirely following discontinuation of the treatment (Sontag 1938). No vomiting or diarrhea was observed during the treatment period. Gastrointestinal effects were not reported in studies examining longer term occupational exposure to white phosphorus (Heimann 1946; Hughes et al. 1962; Kennon and Hallam 1944; Ward 1928). Erosion and hemorrhages in tissue in the esophagus and stomach were observed following ingestion of a fatal unknown quantity of white phosphorus by a cat (Frye and Cucuel 1969). Vomiting was observed in 6 of 21 dogs treated by gavage with an unknown quantity of white phosphorus from firecrackers (Dwyer and Helwig 1925). No gross or microscopic alterations were observed in the gastrointestinal tract of rats treated by gavage with 0.075 mg/kg/day for 204 days (IRDC 1985). Hematological Effects. Hematological effects have been reported in a number of case histories of individuals accidentally or intentionally ingesting a single dose of white phosphorus contained in rat (and cockroach) poisons or fireworks. Because most of the individuals vomited or received gastric lavage shortly after ingestion, the amount of white phosphorus available for absorption is not known. Increases in erythrocyte levels (Diaz-Rivera et al. 1950) and hemoglobin levels (Diaz-Rivera et al. 1950; McIntosh 1927); decreases in erythrocyte levels (Dwyer and Helwig 1925) and hemoglobin and/or hematocrit levels (Simon and Pickering 1973); and anemia (Caley and Kellock 1955) have been observed in some of these individuals. A number of individuals had no change in erythrocyte parameters (Ehrentheil 1957; Fletcher and Galambos 1963; Newburger et al. 1948; Simon and Pickering 1976). The decreases in erythrocyte parameters may be a reflection of the hemorrhages observed in specific tissues (e.g., gastrointestinal tract, liver, skin) (Dathe and Nathan 1946; Hann and Veale 1910; Humphreys and Halpert 1931; Wechsler and Wechsler 1951; Winek et al. 1973). In addition to these changes in erythrocyte parameters, changes in total or differential leukocyte levels were reported in a number of individuals acutely exposed to white phosphorus. Decreases in total leukocyte levels (Diaz-Rivera et al. 1950; Ehrentbeil 1957; Fletcher and Galambos 1963; McCarron et al. 1981; Newburger et al. 1948; Pietras et al. 1968) and decreases or increases in the percentage of polymorphonuclear leukocytes (neutrophils) have been reported (Ehrentheil 1957; McCarron et al. 1981; Pietras et al. 1968). No changes in leukocyte parameters were observed in a number of individuals (Fletcher and Galambos 1963; Newburger et al. 1948; Simon and Pickering 1976). Abnormally low protbrombin times or levels (hypo-prothrombinemia) and a moderate decrease in platelets were observed in a number of individuals ingesting single doses of white phosphorus (Caley and Kellock 1955; Dathe and Nathan 1946; Ehrentheil 1957; Fletcher and Galambos 1963; McCarron et al 1981). Most of the patients developed hypoprothrombinemia within 4-8 days (McCarron et al. 1981). This is probably secondary to the liver damage rather than a direct effect on platelets. No changes in hematological parameters were observed in a child ingesting phosphorized cod liver oil (0.083 mg/kg/day phosphorus) for 184 days (Sontag 1938). Anemia and leukopenia were observed in individuals occupationally exposed to white phosphorus chronically (Ward 1928). It is likely that workers were exposed by the inhalation, oral, and dermal routes. Because there is very little consistency regarding the length of time that elapsed between ingestion and measurement of hematological parameters and the doses cannot be calculated, it is difficult to compare the results of different studies. There is insufficient information to determine whether white phosphorus has a direct effect on erythrocytes and/or leukocytes. The effects observed may be secondary effects. The decrease in erythrocyte, hemoglobin, hematocrit and leukocyte levels may be secondary to hemorrhaging or hematoemesis (Diaz-Rivera et al. 1950; Rubitsky and Myerson 1949) and the increase in erythrocytes and hemoglobin may be a compensatory mechanism due to tissue anoxia. However, since red blood cell synthesis takes 3-5 days, the observed effects may be direct if they are occurring within l-2 days. A slight decrease in hemoglobin levels and increase in eosinophil levels were observed in a 30-year-old man who performed magic shows that involved placing white phosphorus pellets in the mucobuccal folds of his mouth for 15 years. He had no other personal habits that might adversely affect his health except for occasional bidi smoking for about 8 years (Jakhi et al. 1983). Information on hematological effects in animals is limited to one study in which a marked increase in total leukocyte levels and the percentage of monocytes were observed in a guinea pig acutely exposed to 0.9-2.4 mg/kg/day of white phosphorus in a complex dosing regimen (Lawrence and Huffman 1929). The study authors did not specify at which doses the effects occurred. Musculoskeletal Effects. Following ingestion of a fatal dose of rat poison containing white phosphorus by a woman, autopsy revealed fatty infiltration of essentially all tissues, including the musculature (Wertham 1932). Similar effects were reported following the death of a male child who accidentally ingested a firecracker containing white phosphorus; autopsy revealed fatty deposition in many tissues, included the diaphragmic muscle (Humphreys and Halpert 1931). Humans occupationally exposed to white phosphorus probably ingested some airborne white phosphorus. In a study of 71 humans occupationally exposed to white phosphorus, oral exposure to white phosphorus via hand-to-mouth activity was likely because the workers constantly handled a paste containing 4-6% white phosphorus and washroom facilities were inadequate (Ward 1928). In workers exposed to white phosphorus for intermediate durations, 2 of 44 developed phossy jaw, described as slight necrosis in the lower jaw. In workers exposed to white phosphorus for chronic durations, 12 of 27 developed phossy jaw, with necrosis ranging from slight to severe; 2 of the 12 workers developing phossy jaw died from complications related to the necrosis. The progression of the disease was similar in the cases described, usually beginning with the extraction of one or more teeth, poor healing of the socket, followed by necrosis of tissue in the jaw with severe pain and infection. Treatment consisted of repeated removal of destroyed bone tissue and teeth, draining of abscesses, and reconstructive surgery. In severe cases, extensive removal of necrotic bone tissue led to permanent disfigurement. However, exposure levels of white phosphorus were not reported (Ward 1928). Case reports of development of phossy jaw following intermediate or chronic occupational exposure to unreported levels of white phosphorus and phosphorus compounds describe a similar progression of symptoms, with similar results; even in cases of early diagnosis and prompt, intensive treatment of phossy jaw, recovery often took several years (Heimann 1946; Hughes et al. 1962; Kennon and Hallam 1944). It is likely that the effect of white phosphorus in the oral cavity is local, resulting from contact of “inhaled”white phosphorus particles with tissue in the mouth. White phosphorus may affect the oral mucosa. Dull, red spots in the oral mucosa, an early sign of phossy jaw, have been reported to precede its development in occupationally exposed workers (Kennon and Hallam 1944). The oral mucosa of workers exposed to white phosphorus has been described as having a dull, red, unhealthy appearance (Hughes et al. 1962). Exposed bones may be especially susceptible to the irritating affects of white phosphorus. It is not known whether white phosphorus ingested and absorbed into the systemic circulation contributed to the development of phossy jaw. Not all workers exposed to white phosphorus for longer-term durations developed phossy jaw. In a study of 71 workers exposed to airborne white phosphorus for intermediate or chronic durations, 4.5% and 44%, respectively, developed phossy jaw (Ward 1928). Forty-eight male workers with exposure to white phosphorus ranging from 1 to 17 years were found to be normal and healthy with regards to many parameters, including serum levels of calcium and phosphorus, and bone density; none of the men developed phossy jaw (Hughes et al. 1962). Tooth loss often precedes and accompanies the progression of development of phossy jaw (Heimann 1946; Hughes et al. 1962; Kennon and Hallam 1944; Ward 1928). Tooth loss during the later stages of phossy jaw clearly results from destruction of the-bone structure supporting the teeth (Heimann 1946; Hughes et al. 1962; Kennon and Hallam 1944; Legge 1920; Ward 1928). It is not known if tooth loss prior to diagnosis of phossy jaw or early in the development of the condition is related to the white phosphorus exposure. Poor dental hygiene alone can result in tooth loss, and in several case reports some of the workers were described as having poor dental hygiene (Kennon and Hallam 1944). Tooth loss followed by poor healing of the socket often precedes development of the necrosis (Heimann 1946; Hughes et al. 1962; Kennon and Hallam 1944; Ward 1928), suggesting that poor dental hygiene and exposure to white phosphorus may both be contributing factors to the development of phossy jaw. In a case report, five men developed “precursor signs” (delayed healing of extracted tooth sites and residual sepsis) of phossy jaw developed following tooth extraction and occupational exposure to white phosphorus (Hughes et al. 1962). However, the condition did not develop into “classical” phossy jaw. A man was repeatedly exposed to white phosphorus pellets, placed in the right mucobuccal cavity for magic shows, for . 15 years (Jakhi et al. 1983). After . 14.5 years of this type of exposure to white phosphorus, right maxillary molars became loose, and were subsequently lost. This was followed by a lack of healing and development of fistulae in the sockets of the right maxillary molars. White phosphorus necrosis of the jaw developed, with massive necrosis of the maxilla and floor of the antrum on the right side of the mouth; perforations were present through which the maxillary sinus and nasal cavity were visible. No effects were observed on the left side of the maxilla or on the mandible. Radiographs revealed no evidence of pathology in the chest and long bones. The damage to the jaw was probably caused by direct local contact of phosphorus with the soft tissue and bone in the oral cavity. No microscopic or histological abnormalities were observed in the bone of rats treated by gavage with 0.075 mg/kg/day for 204 days (IRDC 1985). Rats exposed for a chronic duration to 0.2 mg/kg/day white phosphorus in the diet had epiphyseal line thickening and greater extension of trabeculae into the diaphysis of unspecified bones, compared to a control group (Fleming et al. 1942). This study is limited by the failure to specify incidences of effects at interval during dosing and by the failure to state the dosing duration explicitly. Bone effects were observed in children (Compere 1930a; Phemister 1918; Sontag 1938) and young animals (Adams 1938a, 1938b; Adams and Sarnat 1940; Whalen et al. 1973) following acute and intermediate oral exposure to white phosphorus. Because white phosphorus-related effects were observed in growing bones, these effects were considered developmental effects, and are discussed in. Section 2.2.2.6. Hepatic Effects. Hepatic effects have been observed in most individuals accidentally or intentionally ingesting a single dose of white phosphorus. These effects include jaundice (Caley and Kellock 1955; Diaz-Rivera et al. 1950, 1961; Ehrentheil 1957; Greenberger et al. 1964; Humphreys and Halpert 1931; McCarron et al. 1981), hepatomegaly (Diaz-Rivera et al. 1950; Fletcher and Galambos 1963; Humphreys and Halpert 1931; Rao and Brown 1974; Simon and Pickering 1976; Wechsler and Wechsler 1951), increased levels of serum bilirubin (Caley and Kellock 1955; Fletcher and Galambos 1963; McCarron et al. 1981; Pietras et al. 1968), impaired liver function test results (Fletcher and Galambos 1963; Newburger et al. 1948; Pietras et al. 1968; Rubitsky and Myerson 1949), and increases in AST, ALT, and/or lactate dehydrogenase (Ehrentheil 1957; Matsumoto et al. 1972; McCarron et al. 1981; Pietras et al. 1968). In addition to these effects, autopsies or liver biopsies have revealed a number of histological alterations in these individuals. Necrosis (Fletcher and Galambos 1963; Rao and Brown 1974; Wechsler and Wechsler 1951), degeneration (Dwyer and Helwig 1925; Greenberger et al. 1964; Wechsler and Wechsler 1951), fibrosis (Greenberger et al. 1964), hemorrhages (Wechsler and Wechsler 1951), and fatty infiltration (Dwyer and Helwig 1925; Hann and Veale 1910; Humphreys and Halpert 1931; Wertham 1932) have been observed in the liver. In addition to these effects, altered prothrombin time or level has been observed in a number of individuals ingesting a single dose of white phosphorus (Caley and Kellock 1955; Dathe and Nathan 1946; Ehrentheill957; Fletcher and Galambos 1963; McCarron et al. 1981). Prothrombin and other plasma proteins that are required for the efficient progression and regulation of blood coagulation are primarily synthesized in the liver. A deficiency of these proteins is often observed in individuals with severe liver disease. A prolongation of prothrombin time is in part due to this impaired synthesis. Liver function tests were normal in workers chronically exposed to unreported levels of airborne phosphorus (Hughes et al. 1962). Similar hepatic alterations have been observed in animals acutely exposed to white phosphorus. Increases in AST and ALT levels (Paradisi et al. 1984), impaired liver function tests (Ghoshal et al. 1969; Hurwitz 1972; Sigal et al. 1954) increased liver weight (Ghoshal et al. 1969; Seakins and Robinson 1964), increased hepatic triglyceride levels (Ghoshal et al. 1969; Pani et al. 1972; Paradisi et al. 1984; Seakins and Robinson 1964), decreased protein synthesis (Barker et al. 1963; Seakins and Robinson 1964), disaggregation of polyribosomes (Pani et al. 1972), fatty degeneration (Ghoshal et al. 1969) and necrosis (Ghoshal et al. 1969) have been observed. No NOAEL values for hepatic effects following acute animal exposure were identified. In rats, the LOAEL value for liver effects was 6 mg/kg (Barker et al. 1963); in mice it was 5 mgkglday (Hurwitz 1972); and in dogs it was 0.2 mg/kg/day (Sigal et al. 1954). The liver effects occurred shortly after dosing; 3 hours after dosing, a significant decrease in protein synthesis was observed in the liver (Barker et al. 1963), minimal hepatocytic fatty changes were observed after 4 hours (Ghoshal et al. 1969), and severe hepatocytic fatty changes were observed after 12 hours (Ghoshal et al. 1969). The following hepatic effects have been observed in animals orally exposed for an intermediate duration fatty infiltration in guinea pigs exposed to 0.75 mg/kg/day (Ashbum et al. 1948), presence of eosinophilic granules at 0.25 mg/kg/day and cirrhosis at 0.66 mg/kg/day in rabbits and guinea pigs (Mallory 1933), and fibrosis and cirrhosis in pigs exposed to 0.6 mg/kg for 5 days/week (Peterson et al. 1991). In the Peterson et al. (1991) study, no liver effects were observed after 4 weeks of exposure; after 8 weeks, there were early signs of fibrosis, and after 12 weeks, extensive fibrosis was observed. Exposure to 0.075 mg/kg/day for an intermediate duration resulted in slight-to-moderate liver necrosis in dying pregnant rats (Bio/dynamics 1991), but no hepatic effects in the surviving pregnant rats or in male rats (Bio/dynamics 1991). In another reproduction study, liver effects were not observed in dying pregnant rats exposed to 0.075 mg/kg/day (IRDC 1985). Both studies used similar exposure protocols and similar vehicles; the difference in the occurrence of liver damage between the studies cannot be explained. Renal Effects. Evidence of severe renal effects have been observed in a number of individuals intentionally or accidentally ingesting a single dose of white phosphorus contained in rat (or roach) poison or fireworks. Proteinuria (Matsumoto et al. 1972; Pietras et al. 1968; Rao and Brown 1974), albuminuria (Dathe and Nathan 1946; Diaz-Rivera et al. 1950; Dwyer and Helwig 1925; Fletcher and Galambos 1963; McCarron et al. 1981; Rubitsky and Myerson 1949), acetonuria (Pietras et al. 1968), increased urobilinogen (Matsumoto et al. 1972), oliguria (Dathe and Nathan 1946; McCarron et al. 1981; Rao and Brown 1974), increased blood levels of urea and/or nitrogen (Diaz-Rivera et al. 1950, 1961; McCarron et al. 1981; Newburger et al. 1948; Pietras et al. 1968; Rao and Brown 1974; Rubitsky and Myerson 1949), and increased blood creatinine levels (Dathe and Nathan 1946; McCarron et al. 1981) have been observed in these individuals. Renal insufficiency may be due to a direct toxic effect of phosphorus on the kidneys or to acute renal tubular necrosis from fluid loss and shock. Patients in shock may have a peculiar pallor and cyanosis. These probably reflect extensive cellular damage with poor perfusion of the capillary beds, and are a prognostic sign of serious health effects (Melamon et al. 1981). Several case reports have reported no alterations in kidney function (Ehrentheil 1957; Fletcher and Galambos 1963; Greenberger et al. 1964; Simon and Pickering 1976). Histological alterations have also been observed in a number of humans ingesting a single dose of white phosphorus. Fatty changes in the tubules and loop of Henle (Dwyer and Helwig 1925; Humphreys and Halpert 1931; Wertham 1932) and engorged glomeruli and intratubular capillaries (Wechsler and Wechsler 1951) have been observed. Because most individuals vomited shortly after ingesting the white phosphorus or were lavaged, accurate doses cannot be calculated except for one study (Harm and Veale 1910). Histological alterations in the kidney were observed in an individual ingesting 2 mg/kg/day, but the lesion was not described. Creatinine levels were similar among unexposed workers and workers exposed to white phosphorus for chronic durations (Hughes et al. 1962). In animals, fatty infiltration in the nephron and subcapsular hemorrhages were observed in dogs acutely exposed to an unspecified amount of white phosphorus (Dwyer and Helwig 1925). No renal effects were observed in rats exposed to 0.075 mg/kg/day for an intermediate duration (Bio/dynamics 1991; IRDC 1985). No chronic exposure animal studies examining renal effects were located. Dermal Effects. Transient toxic dermatitis (described as a scalartiniform rash) developed 9 days after a man ingested a near-fatal dose of rat poison (Dathe and Nathan 1946). Edema of eyelids was reported in a 13-month-old child after ingestion of a fatal dose of white phosphorus (Rao and Brown 1974). Subcutaneous hemorrhages were visible in the left foot in a woman after consumption of 3.9 g of rat poison containing 4% phosphorus (Hann and Veale 1910). The woman died 4 days after the initial poisoning. At this time, an enormous subcutaneous hemorrhage was visible below the waist line. In this case report, the woman apparently did not expel (via vomiting) any of the ingested dose. Thus, it is likely that the ingested dose (2 mg/kg) was representative of the effective dose. Scattered blue-green petechiae were observed on the abdomen of a male child following accidental ingestion of a fatal dose of white phosphorus mixed with other ingredients from a firecracker (Humphreys and Halpert 1931). The dose level in this study could not be determined; the firecracker was a red composition of phosphorus mixed with other ingredients and was thought to contain about 10% phosphorus (Humphreys and Halpert 1931). No studies were located regarding dermal effects in animals after oral exposure to white phosphorus. Other Systemic Effects. A number of other systemic effects have been observed in humans ingesting a single dose of white phosphorus. The effects that are observed most consistently are hypoglycemia (Diaz-Rivera et al. 1950; McCarron et al. 1981; McIntosh 1927; Wechsler and Wechsler 1951), an increase in body temperature (mild pyrexia or fever) (Dathe and Nathan 1946; McIntosh 1927), and a decrease in plasma calcium, potassium, and/or sodium levels (Caley and Kellock 1955; McCarron et al. 1981; Rao and Brown 1974). It is unclear whether the fever seen is a symptom of phosphorus poisoning or a result of the treatment involved. In addition to these effects, metabolic acidosis (Rao and Brown 1974), hypothermia (Simon and Pickering 1976), damage to the spleen (Greenberger et al. 1964), ascites (Fletcher and Galambos 1963), fatty infiltration of the pancreas (Humphreys and Halpert 193 l), and necrosis of the adrenal medulla and cortex (Wechsler and Wechsler 1951) have been observed. In a child ingesting 0.083 mg/kg/day white phosphorus for an intermediate duration, decreased appetite, impaired body weight gain, and poor turgor (fullness or tension produced by the fluid content of blood vessels, capillaries, and cells) were observed (Sontag 1938). Serum glucose levels were decreased in workers occupationally exposed to white phosphorus for a chronic duration. It is likely that the workers were exposed by the inhalation, oral, and dermal routes (Ward 1928). In dogs acutely exposed to an unspecified amount of white phosphorus, hypoglycemia was observed (Williamson and Mann 1923). Rats received intermittent exposure to the atmosphere in the furnace room of a phosphorus factory for 14 months (Ruzuddinov and Rys-Uly 1986). Histology of rats killed monthly revealed progressive morphological degeneration of the tongue and oral mucosa of the cheek, gum, and hard palate. Epithelium and connective tissue from different parts of the oral cavity responded similarly to the treatment. Changes in the epithelial layer, observed after only 1 month of exposure, included increases in keratinization and numbers of cell layers, resulting in thickening and hyperkeratosis in the epithelium of the mucosa. Over time, the thickening and hyperkeratosis in the epithelium increased and histological changes were observed in the subepithelial connective tissue base. Eventually, the oral cavity contained areas of thickening of the mucosa from hyperkeratosis and increased epithelial cell layers interspersed with areas of decreased thickness of the epithelial layer due to atrophy, dystrophy, and cellular necrosis. At this time, adverse changes in the subepithelial connective tissue were considered pronounced. These effects occurred in most of the animals exposed to the atmosphere. It is likely that the observed effects of phosphorus on the oral cavity were local rather than systemic, resulting from direct contact of white phosphorus with tissues in the mouth. The study presented essentially no quantitative data, and the types and exposure levels of chemicals in the atmosphere (thought to contain elementary phosphorus and its inorganic compounds) were not reported (Ruzuddinov and Rys-Uly 1986). 2.2 DISCUSSION OF HEALTH EFFECTS BY ROUTE OF EXPOSURE
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【検索用 登録タグ 作S 作り手】 + 目次 目次 特徴 リンク 曲 CD 動画 関連タグ内の更新履歴 コメント 特徴 作り手名:『soramame128』 UTAU「宮坂ミト」の中の人。自身もUTAUを用いてオリジナル曲を制作している。 ユーザー名は「_」。 リンク soramame128のページ(作者サイト) 曲 おま☆かせがないと生きてけない! 木漏れ日/soramame128 作戦前夜 ザクロ/soramame128 CD まだCDが登録されていません。 動画 関連タグ内の更新履歴 + 関連タグ内の更新履歴 関連タグ内の更新履歴 ※「soramame128」「soramame128CD」タグ内で最近編集やコメントのあった記事を新しい方から10件表示しています。 木漏れ日/soramame128 ザクロ/soramame128 おま☆かせがないと生きてけない! 作戦前夜 コメント 名前 コメント
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Tokyo for Free Imperial Palace East Garden This is a garden adjoining the Imperial Palace and a museum. The garden of 210,000m2 first opened to the public in 1968. Here you can enjoy cherry blossoms and azaleas in spring, various rose in early summer and camellias in winter. Tokyo Metropolitan Government (TMG) Built in 1991, has an observatory on the 48th floor (243m high) which commands a panoramic view of metropolitan Tokyo. A number of sculptures are exhibited outside around the buildings. Museum of Future Science and Technology You can learn about science and scientific experiments, and use the library and mini theatre. Sumo Museum It is the official museum of the Sumo association. It houses more than 20,000 historical and memorial items of Sumo Wrestling, such as paintings, books and photos etc. Meguro Parasite logical Museum This is the only museum in the world on parasites. It exhibits various specimens, studies and photos f parasites. Ebisu Beer Museum Once only the brand name of a beer in Japan, Ebisu is now used as the town name because there was a beer factory here in the Meiji period, the predecessor of Sapporo Beer Compnay, currently one of the most popular beer brands in the country. Here you can learn about the history and technology of making beer. It also features a tasting lounge (with small charge). Currency Museum The museum was opened in 1985 by bank of Japan. It contains about 4,000 representative currency items and exhibits the history of Japanese yen. NHK Broadcast Museum This museum is owned by Japan Broadcast Corporation, featuring high-definition broadcast systems, earlier broadcast equipment, and experience studio, video showing room for older TV programs and a library. Amlux This is the showcase by Toyota Motor Corporation, featuring all range of vehicles as well as mini-theatre, products shop, test driving and technology exhibitions. Honda Welcome Plaza This is the showroom of Honda Corporation. Asimo, the world’s first bipedal humanoid robot, developed by Honda, will welcome you as a guide. You can enjoy exhbitions of new car models. Also featuring a mini-theatre and a product shop. TEPCO Electric Energy Museum You can learn about various ways of electricity is generated, the mechanism of electricity in daily lige through games, videos, interactive exhbitions and experiments etc. Sony building / Sony Style Exhibits new products, offering free-lending and trial, as well as free battery charging for Sony products. Panasonic Center It offers the game trial corner and broadband café lounge, as well as company’s new products. Back to Japan
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入力フォーマットはVTKが基本 example.vtk # vtk DataFile Version 2.0 Unstructured Grid Example ASCII DATASET UNSTRUCTURED_GRID POINTS 4 float 0 0 0 1 0 0 0 1 0 1 1 0 CELLS 2 8 3 0 1 2 3 1 2 3 CELL_TYPES 2 5 5 POINT_DATA 4 SCALARS sample_scalars float 1 LOOKUP_TABLE default 1 2 3 4 VECTORS sample_vectors float 1 0 0 0 1 0 0 -1 0 -1 0 0 TENSORS sample_tensors float 1 0 0 0 1 0 0 0 0 1 0 0 0 1 0 0 0 0 1 0 0 0 1 0 0 0 0 1 0 0 0 1 0 0 0 0 これをベースに改変すればおk 要素上に値をもたせることもできるよ 書式に関する説明は以下参照 http //www.geocities.co.jp/SiliconValley-SantaClara/1183/study/ParaView/VTK/VTK.html(やさしい) http //www.vtk.org/VTK/img/file-formats.pdf(むずかしい) ポイントは ファイルパス、ファイル名に日本語禁止 非構造格子は3Dのみ対応(2Dの時はz=0にする) ポイント番号は0スタート 2Dで流線を書くときはLine Souce(Point Souceは点ではなく球) 動画を作るときは連番VTKファイルを作り、*.vtkを開く エンコードはANSIが無難 バグ 64bit版では動画作成ができない模様です(2011.5現在)
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作曲/作詞:みなと(流星P) 歌:初音ミク 言葉はいつも想いを伝えきれなくて もどかしい 自分に嘘をついたって何も変わらない そうでしょ? 確かなモノなどこの世にないというけれど 本当は 心に強く響く音がある君なら 大丈夫 過去に背を向けて未来へと羽ばたく 空を見上げながらを 蹴り上げて 光射すセカイを感じて 今以上高く飛べたなら 理論なんて超えて行くから 胸焦がして刻む鼓動を 高鳴らせて 引き合う何かを隠して傷ついて事を 避けてた でも今は遠く離れた君の気持ちも わかるんだ 繰り返す始まりと終わりの中を 爆音で翔れば 呼吸が止まる 閉ざされたセカイへの鍵は きっと誰もが手にしてるよ 奇跡なんて信じなくても 自分の手でドアを開けば 何かが変わる Fly away to the sky Fiy away to the sky,run forever 光射すセカイを感じて 今以上高く飛べたなら 理論なんて超えて行くから 胸焦がして刻む鼓動が 高鳴る 手を伸ばして掴み取れ 向かい風さえも切り裂いて 空の青さに飲まれぬように 胸焦がして刻む鼓動よ 君に届け
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[NetworkX] 用語集の生成 $ sort org_tr.txt | uniq sort_uniq.txt 原文 TexTra 改訳(用語集へ) algebraicconnectivity approx_clust_coeff appveyor astar bethehessianmatrix bethehessian betweenness_centrality_subset betweenness_centrality betweenness_subset bfs boykovkolmogorov build_docs capacityscaling closeness_centrality conftest current_flow_betweenness_centrality_subset current_flow_betweenness_centrality current_flow_betweenness_subset current_flow_betweenness current_flow_closeness deploy-key deploy_docs dfs dinitz_alg edge_augmentation equitable_coloring erdos_renyi forking_hell graph6 greedy_coloring_with_interchange greedy_coloring heaps hmn internet_as_graphs label_propagation lanl_routes leda linalg local_and_global_consistency lowest_common_ancestors minors misc mis mycielski neighbor_degree p2g parallel_betweenness preflowpush report_functions_without_rst_generated roget run_with_env second_order shorpaths smetric sparse6 sparsifiers steinertree stochastic structuralholes traversal voterank_alg voterank ./doc/_static ./doc/_スタティック ./doc/_templates ./doc/_テンプレート ./doc/release ./doc/リリース ./doc/reference/algorithms ./doc/参照/アルゴリズム ./doc/reference/classes ./doc/参照/クラス ./doc/reference/readwrite ./doc/参照/読み取り/書き込み ./doc/reference ./doc/参照 ./doc/developer/gitwash ./doc/開発者/gitwash ./doc/developer/nxeps/_static ./doc/開発者/nxeps/_スタティック ./doc/developer/nxeps ./doc/開発者/nxeps ./doc/developer ./doc/開発者 ./doc ./doc ./examples/3d_drawing ./examples/3dドローイング ./examples/advanced ./examples/advanced ./examples/applications ./examples/applications ./examples/basic ./examples/basic ./examples/geospatial ./examples/geospatial ./examples/javascript/force ./examples/javascript/force ./examples/javascript ./examples/javascript ./examples/jit ./examples/jit ./examples/pygraphviz ./examples/pygraphviz ./examples/algorithms ./examples/アルゴリズム ./examples/graph ./examples/グラフ ./examples/drawing ./examples/ドローイング ./networkx/linalg/tests ./networkx/linalg/テスト ./networkx/linalg ./networkx/linalg ./networkx/algorithms/shorpaths/tests ./networkx/アルゴリズム/shorpaths/テスト ./networkx/algorithms/shorpaths ./networkx/アルゴリズム/shorpaths ./networkx/algorithms/traversal/tests ./networkx/アルゴリズム/traversal(横断?)/テスト ./networkx/algorithms/traversal ./networkx/アルゴリズム/traversal ./networkx/algorithms/coloring/tests ./networkx/アルゴリズム/カラーリング/テスト ./networkx/algorithms/community/tests ./networkx/アルゴリズム/コミュニティ/テスト ./networkx/algorithms/community ./networkx/アルゴリズム/コミュニティ ./networkx/algorithms/components ./networkx/アルゴリズム/コンポーネント ./networkx/algorithms/tree/tests ./networkx/アルゴリズム/ツリー/テスト ./networkx/algorithms/tree ./networkx/アルゴリズム/ツリー ./networkx/algorithms/tests ./networkx/アルゴリズム/テスト ./networkx/algorithms/node_classification/tests ./networkx/アルゴリズム/ノード分類/テスト ./networkx/algorithms/node_classification ./networkx/アルゴリズム/ノード分類 ./networkx/algorithms/flow/tests ./networkx/アルゴリズム/フロー/テスト ./networkx/algorithms/flow ./networkx/アルゴリズム/フロー ./networkx/algorithms/link_analysis/tests ./networkx/アルゴリズム/リンク解析/テスト ./networkx/algorithms/link_analysis ./networkx/アルゴリズム/リンク解析 ./networkx/algorithms/centrality/tests ./networkx/アルゴリズム/中心性/テスト ./networkx/algorithms/centrality ./networkx/アルゴリズム/中心性 ./networkx/algorithms/bipartite/tests ./networkx/アルゴリズム/二部(bipartite)/テスト ./networkx/algorithms/bipartite ./networkx/アルゴリズム/二部(bipartite) ./networkx/algorithms/isomorphism/tests ./networkx/アルゴリズム/同形性/テスト ./networkx/algorithms/isomorphism ./networkx/アルゴリズム/同形性 ./networkx/algorithms/assortativity/tests ./networkx/アルゴリズム/同類性/テスト ./networkx/algorithms/assortativity ./networkx/アルゴリズム/同類性 ./networkx/algorithms/connectivity/tests ./networkx/アルゴリズム/接続性/テスト ./networkx/algorithms/connectivity ./networkx/アルゴリズム/接続性 ./networkx/algorithms/components/tests ./networkx/アルゴリズム/構成部品/テスト ./networkx/algorithms/operators/tests ./networkx/アルゴリズム/演算子/テスト ./networkx/algorithms/operators ./networkx/アルゴリズム/演算子 ./networkx/algorithms/approximation/tests ./networkx/アルゴリズム/近似/テスト ./networkx/algorithms/approximation ./networkx/アルゴリズム/近似 ./networkx/algorithms ./networkx/アルゴリズム ./networkx/classes ./networkx/クラス ./networkx/generators/tests ./networkx/ジェネレータ/テスト ./networkx/generators ./networkx/ジェネレータ ./networkx/testing/tests ./networkx/テスト/テスト ./networkx/testing ./networkx/テスト ./networkx/tests ./networkx/テスト ./networkx/drawing ./networkx/ドローイング ./networkx/utils/tests ./networkx/ユーティリティ/テスト ./networkx/utils ./networkx/ユーティリティ ./networkx/readwrite/json_graph/tests ./networkx/読み取り/書き込み/JSON_graph/テスト ./networkx/readwrite/tests ./networkx/読み取り/書き込み/テスト ./networkx ./networkx ./tools/appveyor ./tools/appveyor ./tools/travis ./tools/travis ./tools ./tools ./examples/subclass ./例/サブクラス ./examples ./例 ./requirements ./要件 . . ./networkx/readwrite/json_graph /.networkx/読み取り/書き込み/JSON_graph ./networkx/algorithms/coloring /networkx/アルゴリズム/カラーリング ./networkx/classes/tests /networkx/クラス/テスト ./networkx/drawing/tests /networkx/ドローイング/テスト ./networkx/readwrite /networkx/読み取り/書き込み 3d_drawing 3dドローイング four_grids 4グリッド api_0.99 API_0.99 api_1.0 API_1.0 api_1.10 API_1.10 api_1.11 API_1.11 api_1.4 API_1.4 api_1.5 API_1.5 api_1.6 API_1.6 api_1.7 API_1.7 api_1.8 API_1.8 api_1.9 API_1.9 cytoscape Cytoscape dag DAG(ダグ) gml GML git_intro Git_intro git_install Git_インストール git_resources Git_リソース git_links Git_リンク git_development Git_開発 graphml GraphML json_graph JSON_graph javascript Java Script linux_install Linux インストール Makefile Makefile networkx NetworkX networkx_logo NetworkX_ロゴ unix_email UNIX_email yaml YAML __init__ __init__ _static _スタティック _templates _テンプレート adjlist adjリスト agraph agraph all_random_functions all_random_関数 antigraph antigraph attrmatrix attrマトリックス conf conf d_separation d_分割 dense_numpy dense_NumPy dev_banner dev_バナー doc doc eol_banner eol_バナー geopandas geopandas gexf gexf gitwash gitwash gitwash_dumper gitwashダンパー gl1.gpickle gl1.gpickle gpickle gpickle gw1.gpickle gw1.gpickle ismags ismags iso_r01_s80 iso_r01_s80 isomorphvf2 isomorphvf2 jit jit krackhardt_centrality krackhardt_中心性 kcutsets kカットセット kclique kクリーク kcomponents kコンポーネント lgc lgc mapped_queue mapped_queue mayavi2_spring mayavi2_スプリング migration_guide_from_1.x_to_2.0 migration_guide_from_1.x_to_2.0 mst mst netgen-2.gpickle netgen-2.gpickle networksimplex networksimplex nx_yaml nx_YAML nx_agraph nx_agraph nx_pydot nx_pydot nx_pylab nx_pylab nx_shp nx_shp nxep-0000 nxep-0000 nxep-0001 nxep-0001 nxep-0002 nxep-0002 nxep-template nxep-テンプレート nxeps nxeps osmnx osmnx printgraph printgraph pydot pydot pygraphviz pygraphviz pygraphviz_simple pygraphviz_シンプル pygraphviz_attributes pygraphviz_属性 pygraphviz_draw pygraphviz_描画 pylab pylab pysal pysal rcm rcm rgraph rgraph roget_dat.txt roget_dat.txt shortestaugmentingpath shortestaugmentingpath shp shp si2_b06_m200 si2_b06_m200 smetric smetric sparse6 sparse6 sparsegraph6 sparsegraph6 temporalisomorphvf2 temporalisomorphvf2 vf2userfunc vf2userfunc wlm3.gpickle wlm3.gpickle words_dat.txt words_dat.txt write_dotfile write_dotfile README お読みください this_project このプロジェクト threshold しきい値 all すべて introduction はじめに atlas アトラス atlas.dat アトラス.dat applications アプリケーション algorithms アルゴリズム INSTALL インストール install インストール before_install インストールの前 interval_graph インターバル・グラフ index インデックス independent_set インデペンデント・セット wiener ウィーナー extra エクストラ expanders エクスパンダ ego エゴ ego_graph エゴグラフ edgebfs エッジbfs edgedfs エッジdfs edgelist エッジリスト edge_kcomponents エッジ・kコンポーネント edge_colormap エッジ・カラーマップ edmondskarp エドモンズカープ euler オイラー(euler) operations オペレーション custom カスタム katz カッツ katz_centrality カッツ中心性 cuts カット covering カバー coloring カラーリング kernighan_lin カーニハン・リン quality クオリティ knuth_miles クヌース・マイル knuth_miles.txt クヌース・マイル.txt classic クラシック classes クラス clustering クラスタリング clustering_coefficient クラスタリング係数 cluster クラスター clique クリーク credits クレジット graph グラフ graph_historical グラフ_歴史的 graphical グラフィック graphviews グラフビュー graph_hashing グラフ・ハッシュ graphmatrix グラフ・マトリックス group グループ core コア coreviews コアビュー core_developer コア開発者 cographs コグラフ copybutton コピーボタン community コミュニティ community_utils コミュニティ・ユーティリティ contextmanager コンテクスト・マネージャ contextmanagers コンテクスト・マネージャー components コンポーネント(構成要素) component コンポーネント(構成要素) coding コーディング gomory_hu ゴモーリー・フー cycles サイクル subclass サブクラス sampson サンプソン sampson_data サンプソン・データ circular_tree サークルツリー simple_graph シンプルグラフ simple_paths シンプルパス simple_path シンプルパス generators ジェネレータ geometric ジオメトリック(幾何学的) giant_component ジャイアントコンポーネント joint_degree_seq ジョイント次数_seq stoer_wagner ストアー・ワグナー stoerwagner ストアー・ワグナー spectral_graph_forge スペクトラルグラフ・フォージ spectrum スペクトル spectral_bipartivity スペクトル_bipartivity spectral スペクトルの spectral_grid スペクトル・グリッド special スペシャル small スモール smallworld スモールワールド swap スワップ setup セットアップ set_up_fork セットアップ・フォーク social ソーシャル chess_masters チェス・マスター chess_masters_WCC.pgn チェス・マスター_WCC.pgn chains チェーン tutorial チュートリアル team チーム team_list チームリスト trees ツリー tree ツリー tree_isomorphism ツリー同形性 treewidth ツリー幅 tools ツール testing テスト tests テスト test テスト disjoint_paths ディスジョイント・パス davis_club デイビスクラブ decorators デコレータ default デフォルト travis トラヴィス tournament トーナメント dot_atlas ドットアトラス drawing ドローイング napoleon_russian_campaign ナポレオン・ロシアン・キャンペーン news ニュース node_link ノードリンク node_colormap ノード・カラーマップ node_classification ノード分類 hybrid ハイブリッド harary_graph ハラリグラフ hartford_drug ハートフォード・ドラッグ harmonic ハーモニック harmonic_centrality ハーモニック中心性 harmonic_function ハーモニック関数 vitality バイタリティ binary バイナリ pajek パジェク(pajek) patching パッチ適用 percolation パーコレーション percolation_centrality パーコレーション中心性 historical_tests ヒストリカル・テスト hits_alg ヒット_alg hits ヒット数 beam_search ビームサーチ beamsearch ビームサーチ filters フィルタ forking_button フォーキング・ボタン force フォース(力) football フットボール flow フロー flow_matrix フロー行列 branch_dropdown ブランチ・ドロップダウン bridges ブリッジ blockmodel ブロックモデル pull_button プルボタン project プロジェクト properties プロパティ heavy_metal_umlaut ヘビーメタル・ウムラウト base_test ベース・テスト pairs ペア pagerank ページランク pagerank_alg ページランク_alg voronoi ボロノイ(voronoi) maxflow マックスフロー maxflow_large_graph マックスフロー・ラージ・グラフ matching マッチング match_helpers マッチ・ヘルパー matchhelpers マッチ・ヘルパー MANIFEST マニフェスト multigraph マルチグラフ multiline_adjlist マルチライン_adjリスト multidigraph マルチ有向グラフ maintainer_workflow メンテナ・ワークフロー modularitymatrix モジュラリティ・マトリックス modularity_max モジュラリティ最大 modularity モジュール方式 union_find ユニオン・ファインド unionfind ユニオン・ファインド utils ユーティリティ LICENSE ライセンス license ライセンス laplacian ラプラシアン laplacianmatrix ラプラシアン・マトリックス labels_and_colors ラベルとカラー ramsey ラムゼー random_graphs ランダムグラフ random_sequence ランダムシーケンス random_clustered ランダム・クラスター random_geometric_graph ランダム・ジオメトリック・グラフ randomness ランダム性 rich_club リッチクラブ richclub リッチクラブ reference リファレンス release リリース release_2.0 リリース_2.0 release_2.1 リリース_2.1 release_2.2 リリース_2.2 release_2.3 リリース_2.3 release_2.4 リリース_2.4 release_2.5 リリース_2.5 release_dev リリース_dev release_template リリース・テンプレート links リンク link_prediction リンク予測 link_analysis リンク解析 lukes ルクス layout レイアウト rainbow_coloring レインボー・カラーリング regular レギュラー reportviews レポートビュー roadmap ロードマップ load_centrality ロード中心性 words ワード generic 一般的な triads 三つ組(triads) centrality 中心性 expected_degree_sequence 予想される次数シーケンス second_order_centrality 二次中心性 bipartite 二部(bipartite) intersection 交点 algebraic_connectivity 代数的接続性 examples 例 example 例 example-requirements 例-要件 exceptions 例外 exception 例外 values 値(バリュー) dominance 優性 relabel 再ラベル付け(relabel) redundancy 冗長性 branchings 分岐 dispersion 分散 decomposition 分解 isolates 分離 isolate 分離する reaching 到達する efficiency 効率性 efficiency_measures 効率測定 semiconnected 半連結の unary 単項の bibliography 参考文献 iterated_dynamical_systems 反復力学系 isomorph 同形 isomorphism.ismags 同形.ismags isomorphism 同形性 isomorphism.vf2 同形性.vf2 assortativity 同類性 circuits 回路 eigenvector 固有ベクトル eigenvector_centrality 固有ベクトル中心性 eigenvalues 固有値 geospatial 地理空間 basic 基本 boundary 境界 convert 変換 convert_numpy 変換_NumPy convert_scipy 変換_SciPy convert_pandas 変換_pandas convert_matrix 変換行列 multipartite_graph 多区分グラフ betweenness 媒介性 dense 密な projection 射影 breadth_first_search 幅優先検索 planar_drawing 平面図 planarity 平面性 citing 引用 chordal 弦(chordal) weakly_connected 弱く接続された strongly_connected 強く接続された advanced 拡張 directed 指示された biconnected 接続された connected 接続された connectivity 接続性 dominating 支配的 dominating_set 支配的セット sudoku 数独 known_projects 既知のプロジェクト max_weight_clique 最大重量クリーク mincost 最小コスト digraph 有向グラフ digraph_historical 有向グラフ_歴史的 trophic 栄養の lattice 格子 following_latest 次の最新 degree 次数 degree_alg 次数_alg degree_seq 次数_seq degree_sequence 次数シークエンス degree_centrality 次数中心性 depth_first_search 深さ優先検索 mixing 混合 operators 演算子 glossary 用語集 reciprocity 相反関係 correlation 相関関係 product 積 karate_club 空手クラブ line 線分 dedensification 脱-ち密化 house_with_colors 色付きハウス load 荷重 matrix 行列 CODE_OF_CONDUCT 行動規範 code_of_conduct 行動規範 requirements 要件 summarization 要約 configure_git 設定_Git recognition 認識 attracting 誘引する read_write 読み取り/書き込み readwrite 読み取り/書き込み CONTRIBUTING 貢献 contribs 貢献 contribute 貢献する distance_measures 距離_メジャー distance_regular 距離_レギュラー closeness 近さ approximation 近似 communicability 通信可能性 communicability_alg 通信可能性_alg moral 道徳的 subgraph 部分グラフ subgraph_alg 部分グラフ_alg subgraphviews 部分グラフビュー unweighted 重みなし weighted 重み付き weighted_graph 重み付きグラフ duplication 重複 development_workflow 開発ワークフロー developer 開発者 functions 関数 function 関数 hierarchy 階層 adjacency 隣接関係 asteroidal 非ステロイド系 non_randomness 非ランダム性 nonisomorphic_trees 非同形ツリー asyn_fluid 非同期流体 deprecations 非推奨 vertex_cover 頂点カバー ordered 順序づけられた similarity 類似性 $ sort org_tr.txt | uniq sort_uniq.txt およそ580行。うち80行ほどがパス行。 500項目程度の対訳が得られる。 $ sort -t '|' -k 2 sort_uniq.txt sort_uniq_sort.txt 未翻訳が70行ほど。 (500 - 70) ÷ 500 = 0.86 翻訳率、86%の出来。 [正誤は未確認。文脈内容も未確認] これを NetworkX_1st 用語集として TexTra 登録する。 ファイル名にもちいられた用語ということは、本文でも頻出することが予想される。逆に、これらを訳せないと本文の翻訳率も期待できない。 今後は、本文 po の翻訳を開始。 1st 用語集の修正 2nd 用語集の準備、登録 翻訳後の sed リプレース(一括置換) 翻訳 po の TexTra 対訳集への登録。 2021年6月23日 公開 2021年6月26日 更新 しだひろし/PoorBook G3'99 リンク・引用・転載は自由です。 カウンタ: - 名前 コメント
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https://w.atwiki.jp/touhoukashi/pages/478.html
【登録タグ RegaSound V Vivid Parasol らむだーじゃん ネクロファンタジア 曲】 【注意】 現在、このページはJavaScriptの利用が一時制限されています。この表示状態ではトラック情報が正しく表示されません。 この問題は、以下のいずれかが原因となっています。 ページがAMP表示となっている ウィキ内検索からページを表示している これを解決するには、こちらをクリックし、ページを通常表示にしてください。 /** General styling **/ @font-face { font-family Noto Sans JP ; font-display swap; font-style normal; font-weight 350; src url(https //img.atwikiimg.com/www31.atwiki.jp/touhoukashi/attach/2972/10/NotoSansCJKjp-DemiLight.woff2) format( woff2 ), url(https //img.atwikiimg.com/www31.atwiki.jp/touhoukashi/attach/2972/9/NotoSansCJKjp-DemiLight.woff) format( woff ), url(https //img.atwikiimg.com/www31.atwiki.jp/touhoukashi/attach/2972/8/NotoSansCJKjp-DemiLight.ttf) format( truetype ); } @font-face { font-family Noto Sans JP ; font-display swap; font-style normal; font-weight bold; src url(https //img.atwikiimg.com/www31.atwiki.jp/touhoukashi/attach/2972/13/NotoSansCJKjp-Medium.woff2) format( woff2 ), url(https //img.atwikiimg.com/www31.atwiki.jp/touhoukashi/attach/2972/12/NotoSansCJKjp-Medium.woff) format( woff ), url(https //img.atwikiimg.com/www31.atwiki.jp/touhoukashi/attach/2972/11/NotoSansCJKjp-Medium.ttf) format( truetype ); } rt { font-family Arial, Verdana, Helvetica, sans-serif; } /** Main table styling **/ #trackinfo, #lyrics { font-family Noto Sans JP , sans-serif; font-weight 350; } .track_number { font-family Rockwell; font-weight bold; } .track_number after { content . ; } #track_args, .amp_text { display none; } #trackinfo { position relative; float right; margin 0 0 1em 1em; padding 0.3em; width 320px; border-collapse separate; border-radius 5px; border-spacing 0; background-color #F9F9F9; font-size 90%; line-height 1.4em; } #trackinfo th { white-space nowrap; } #trackinfo th, #trackinfo td { border none !important; } #trackinfo thead th { background-color #D8D8D8; box-shadow 0 -3px #F9F9F9 inset; padding 4px 2.5em 7px; white-space normal; font-size 120%; text-align center; } .trackrow { background-color #F0F0F0; box-shadow 0 2px #F9F9F9 inset, 0 -2px #F9F9F9 inset; } #trackinfo td ul { margin 0; padding 0; list-style none; } #trackinfo li { line-height 16px; } #trackinfo li nth-of-type(n+2) { margin-top 6px; 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} /** Infomation styling **/ #trackinfo .info_header th { padding .3em .5em; background-color #D8D8D8; font-size 95%; } #trackinfo .infomation_show_btn_wrapper { float right; font-size 12px; user-select none; } #trackinfo .infomation_show_btn { cursor pointer; } #trackinfo .info_content td { padding 0 0 0 5px; height 0; transition .3s; } #trackinfo .info_content ul { padding 0; margin 0; max-height 0; list-style initial; transition .3s; } #trackinfo .info_content li { opacity 0; visibility hidden; margin 0 0 0 1.5em; transition .3s, opacity .2s; } #trackinfo .info_content.infomation_show td { padding 5px; height 100%; } #trackinfo .info_content.infomation_show ul { padding 5px 0; max-height 50em; } #trackinfo .info_content.infomation_show li { opacity 1; visibility visible; } #trackinfo .info_content.infomation_show li nth-of-type(n+2) { margin-top 10px; } /** Lyrics styling **/ #lyrics { font-size 1.06em; line-height 1.6em; } .not_in_card, .inaudible { display inline; position relative; 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